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Dry Eye
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I always suspected that the pressure issues in glaucoma were secondary; as is evidenced by “normal pressure” glaucoma, where the same damage to the retina is seen. The retina is one of the most chemically-active tissues, having to concentrate light and then process it. It makes sense that it would be particularly vulnerable to different metabolic stressors. In addition, it also makes sense that since metabolic stressors can be different, the type of damage could be different. I believe this applies to glaucoma and macular degeneration. Just as with high- and low-pressure glaucoma, there is wet and dry macular degeneration. Do these labels/categories really apply or are they all variants of an underlying root cause? We could also include cataracts, where the damage from the sun affects the lens separately from the retina. There is good data on the mechanism of cataract and potential modulators/remedies.
Back to glaucoma, I’ve collected some recent articles that support this idea and suggest that one of my core nutients, niacin, may have a role in its prevention and even possible reversal.
References (start at the top)
Crosstalk Between Dysfunctional Mitochondria and Inflammation in Glaucomatous Neurodegeneration.2021
Nicking-Glaucoma-with-Nicotinamide.2017.nejm
Glaucoma as a Metabolic Optic Neuropathy–Making the Case for Nicotinamide Treatment in Glaucoma
!Bone Lead Levels and Risk of Incident Primary Open-Angle Glaucoma–The VA Normative Aging Study.2018
Diurnal and 24-h Intraocular Pressures in Glaucoma
Medical Management of Glaucoma in the 21st Century from a Canadian Perspective.2016
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H Pylori and Eye Disease
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Immune, Allergy & Infection Program
This is an amazing set of articles and discovery about the relationship between an infection (H Pylori), typically implicated in stomach-acid-reflux conditions and eye health! Shows that these organisms haven’t studied anatomy and go whereever they please!
References (start at the top)
Helicobacter pylori Infection and Eye Diseases– A Systematic Review.2014
Floaters
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Posterior Vitreous Detachment
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The eye develops by the merging of the front and back of the eye. The back of the eye, the retina (and its related tissues) begins as an outpocketing of the brain into the eye socket. The front of the eye, which forms the cornea, lens and vitreous body forms from other tissues (ectoderm) which then merges with the back. Along this “seam” is where detachments occur (retinal and posterior vitreous).
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Retinal Detachment
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The eye develops by the merging of the front and back of the eye. The back of the eye, the retina (and its related tissues) begins as an outpocketing of the brain into the eye socket. The front of the eye, which forms the cornea, lens and vitreous body forms from other tissues (ectoderm) which then merges with the back. Along this “seam” is where detachments occur (retinal and posterior vitreous).
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Conjunctivitis
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Uveitis
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Uveitis is inflammation of the uvea. The uvea is the middle layer of tissue in the wall of the eye. It consists of the iris, the ciliary body and the choroid.
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Night Blindness
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